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Activating FGFR3 mutations cause mild hyperplasia in human skin, but are insufficient to drive benign or malignant skin tumors.

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Cell Cycle2014PMID 24626198PMC4050160stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

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Linked entities

Linked entities (3)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 3

Curated evidence

Evidence citing this paper (2)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
24626198
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–2 of 2 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
FGFR3 G697C1
(oncogenic)Malignant NeoplasmALIASOncogenicDDoes Not Support Oncogenicity3accepted
EID10838

In human skin grafts, FGFR3, R248C, and S249C, but not G697C induced MAPK activation and hyperproliferation. The authors conclude that G697C is likely not an oncodriver and may be a geographically ass… (full text at CIViC)

PMID 24626198 · Duperret et al., 2014 · Open in CIViC

civic
FGFR3 R248C1
(oncogenic)Squamous Cell CarcinomaOncogenicDSupports Oncogenicity3submitted
EID8320

Expression of FGFR3 carrying the p.Arg248Cys variant in HEK293 cells resulted in increased phospho-ERK, indicating activation of the MAP Kinase pathway. Expression of FGFR3 p.Arg248Cys in a human kera… (full text at CIViC)

PMID 24626198 · Duperret et al., 2014 · Open in CIViC

civic