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Differentiation of NUT midline carcinoma by epigenomic reprogramming.

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Cancer Res2011PMID 21447744PMC3070805stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (4)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 4

Curated evidence

Evidence citing this paper (2)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
21447744
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–2 of 2 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
NUTM1 Fusion2
PanobinostatNUT CarcinomaALIASPredictiveDSupports Sensitivity Response3accepted
EID12099

BRD4-NUT fusion was found to be associated with globally decreased histone acetylation and transcriptional repression in gain-of-expression and loss-of-expression assays. Following these results, it w… (full text at CIViC)

PMID 21447744 · Schwartz et al., 2011 · Open in CIViC

civic
VorinostatNUT CarcinomaALIASPredictiveCSupports Sensitivity Response3submitted
EID12100

A 10-year-old male patient with NUT Midline Carcinoma (NMC) harbouring the BRD4-NUT fusion gene was treated with vorinostat, an HDAC inhibitor, at a daily dose of 400 mg. In patient-derived NMC cells,… (full text at CIViC)

PMID 21447744 · Schwartz et al., 2011 · Open in CIViC

civic