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MMP1-induced NF-κB activation promotes epithelial-mesenchymal transition and sacituzumab govitecan resistance in hormone receptor-positive breast cancer.

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Cell Death Dis2025PMID 40287412PMC12033297stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (3)

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Validated 3

Curated evidence

Evidence citing this paper (1)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
40287412
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–1 of 1 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
MMP1 Overexpression1
Sacituzumab GovitecanEstrogen Receptor-positive Breast CancerUNRESOLVEDPredictiveDSupports Resistance3accepted
EID12597

In the TIMER dataset, levels of MMP1 is increased in cancers compared with normal tissue, where its levels in sacituzumab govitecan (SG)-resistant tumours are higher than SG-sensitive tumours by IHC. … (full text at CIViC)

PMID 40287412 · Chen et al., 2025 · Open in CIViC

civic