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Alternative transcription initiation leads to expression of a novel ALK isoform in cancer.

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Nature2015PMID 26444240PMC4807020stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (4)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 4

Curated evidence

Evidence citing this paper (2)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
26444240
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–2 of 2 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
ALK Alternative Transcript (ATI)2
CrizotinibCutaneous MelanomaALIASPredictiveCSupports Sensitivity Response4accepted
EID1936

Expression of an alternative ALK transcript (ATI), encompassing exons 20–29 preceded by ~400 base pairs (bp) of intron 19, but not exons 1–19 was identified in three samples of thyroid cancer and mela… (full text at CIViC)

PMID 26444240 · Wiesner et al., 2015 · Open in CIViC

civic
〃Malignant NeoplasmALIASPredictiveDSupports Sensitivity Response4submitted
EID7646

Expression of the ALK alternative transcript isoform (ATI) in Ba/F3 cells resulted in phosphorylation of ALK and IL3-independent growth in vitro, while ALK ATI expressing NIH-3T3 cells induced tumorig… (full text at CIViC)

PMID 26444240 · Wiesner et al., 2015 · Open in CIViC

civic