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NUP98/NSD1 and FLT3/ITD coexpression is more prevalent in younger AML patients and leads to induction failure: a COG and SWOG report.

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Blood2014PMID 25145343PMC4192751stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

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Linked entities

Linked entities (3)

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Validated 3

Curated evidence

Evidence citing this paper (2)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
25145343
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–2 of 2 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
NSD1 Fusion1
(prognostic)Acute Myeloid LeukemiaCURATED_BROADERPrognosticBSupports Poor Outcome3submitted
EID7054

This publication examines coexpression of NUP98/NSD1 and FLT3/ITD in AML, remission, and survival rate in 1421 patients. The CR rate for patients with and without FLT3/ITD was 63% and 82%, respectivel… (full text at CIViC)

PMID 25145343 · Ostronoff et al., 2014 · Open in CIViC

civic
NUP98 Fusion1
(diagnostic)Acute Myeloid Leukemia with NUP98 RearrangementDiagnosticBSupports Positive4accepted
EID12379

In this 2014 study the authors investigated the presence of NUP98::NSD1 fusion in pediatric and adult acute myeloid leukemia (AML) patients. Utilizing reverse transcriptase-polymerase chain reaction (… (full text at CIViC)

PMID 25145343 · Ostronoff et al., 2014 · Open in CIViC

civic