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Emergence of KRAS mutations and acquired resistance to anti-EGFR therapy in colorectal cancer.

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Nature2012PMID 22722830PMC3927413stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (6)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 6

Curated evidence

Evidence citing this paper (5)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
22722830
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–5 of 5 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
KRAS A146T1
CetuximabMalignant Colorectal NeoplasmCURATED_BROADERPredictiveDSupports Resistance3submitted
EID7176

identified a KRAS(A146T) mutation after four passages in increasing concentrations of cetuximab (20 nM and higher; These cells were indeed resistant to the drug. These mutations that arise are the res… (full text at CIViC)

PMID 22722830 · Misale et al., 2012 · Open in CIViC

civic
KRAS G12R1
CetuximabMalignant Colorectal NeoplasmCURATED_BROADERPredictiveDSupports Resistance4accepted
EID3989

In an in vitro study to identify cetuximab-resistant clones following continuous drug treatment, a resistant clone of the Lim1215 colorectal cancer cell line was identified to have the KRAS G12R mutat… (full text at CIViC)

PMID 22722830 · Misale et al., 2012 · Open in CIViC

civic
KRAS G13D1
CetuximabMalignant Colorectal NeoplasmCURATED_BROADERPredictiveDSupports Resistance—accepted
EID3878

In an in vitro study, a Lim1215 colorectal cancer cell line endogenously expressing KRAS G13D mutation demonstrated resistance to cetuximab treatment, compared to Lim1215 parental cells expressing wil… (full text at CIViC)

PMID 22722830 · Misale et al., 2012 · Open in CIViC

civic
KRAS Mutation1
CetuximabMalignant Colorectal NeoplasmCURATED_BROADERPredictiveBSupports Resistance3submitted
EID6464

This study shows that KRAS mutations such as G12D, G13D and KRAS amplification are frequent drivers of acquired resistance to cetuximab in colorectal cancers. KRAS mutant clones can be detected throug… (full text at CIViC)

PMID 22722830 · Misale et al., 2012 · Open in CIViC

civic
KRAS Q61H1
CetuximabMalignant Colorectal NeoplasmCURATED_BROADERPredictiveCSupports Resistance—accepted
EID3862

In a colorectal cancer patient with a KRAS Q61H mutation, KRAS Q61H was reported to be refractory to cetuximab treatment. The patient was treated with cetuximab and irinotecan for 18 months, had a par… (full text at CIViC)

PMID 22722830 · Misale et al., 2012 · Open in CIViC

civic