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Publication

Androgen receptor splice variants drive castration-resistant prostate cancer metastasis by activating distinct transcriptional programs.

Authors not recorded

J Clin Invest2024PMID 38687617PMC11142739stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (3)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 3

Curated evidence

Evidence citing this paper (1)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
38687617
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–1 of 1 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
AR AR-V71
(oncogenic)Malignant Prostate NeoplasmOncogenicDSupports Oncogenicity4accepted
EID12465

To compare the activity of AR-V7 with that of full-length AR (AR-FL) lentiviral stable cell lines of castrate-resistant prostate cancer were generated that expressed doxycycline-regulated AR-V7 or AR-… (full text at CIViC)

PMID 38687617 · Han et al., 2024 · Open in CIViC

civic