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Profound MEK inhibitor response in a cutaneous melanoma harboring a GOLGA4-RAF1 fusion.

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J Clin Invest2019PMID 30835257PMC6486352stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (5)

How each link was made (MeSH, dictionary, registry reference, curation…) and whether it has been validated. Candidate links are not counted in entity statistics.

Validated 5

Curated evidence

Evidence citing this paper (1)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
30835257
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–1 of 1 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
RAF1 Fusion1
Cobimetinib + TrametinibSubstitutesMelanomaPredictiveCSupports Sensitivity Response5accepted
EID8374

In a case study of a metastatic melanoma with a GOLGA4-RAF1 fusion, the patient was administered MEK inhibitor therapy in the absence of other actionable variants. This resulted in a profound and clin… (full text at CIViC)

PMID 30835257 · McEvoy et al., 2019 · Open in CIViC

civic