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SMAD2, SMAD3 and SMAD4 mutations in colorectal cancer.

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Cancer Res2013PMID 23139211stubpubmedProvenance
Source
PubMed
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-CIVIC-20260908-000001
Published

Abstract

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Linked entities

Linked entities (2)

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Validated 2

Curated evidence

Evidence citing this paper (1)

civicProvenance
Source
CIViC — Clinical Interpretation of Variants in Cancer
Dataset
CIViC evidence items
Version
civic-2026-09-08
Retrieved
Sep 8, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Evidence
expert curation
License
CC0 1.0
PMID
23139211
Run
ING-CIVIC-20260908-000001
Open at source
CuratedShowing 1–1 of 1 evidence items · levels, directions and significance as curated at the source; each row links to its CIViC record.
TherapyCancerTypeLevelDirection · significanceRating (1–5)StatusEvidenceSource
SMAD2 S276L1
(functional)—UNRESOLVEDFunctionalDSupports Loss Of Function1accepted
EID11039

SMAD2 S276L mutations were observed in 3/744 cases of colorectal cancer. This mutation was introduced by site-directed mutagenesis and transfected into HEK293T cells. The interaction of wildtype and m… (full text at CIViC)

PMID 23139211 · Fleming et al., 2013 · Open in CIViC

civic