Publication
Phosphorylated HPV‑16 E7 disrupts planar cell polarity in cervical cancer cells through vangl1 dysregulation.
Ifeoluwa D Gbala, Om Basukala, Amira Zine El Abidine, Michael P Myers, Rebecca Bertolio, Giannino Del Sal, Lawrence Banks
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- PubMed
- Retrieved
- Sep 26, 2026
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- normalized (units and labels harmonized; values unchanged)
- Run
- ING-PUBMED-20260926-000001
Abstract
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High‑risk (HR) human papillomaviruses (HPVs) rely on the continuous expression of the E6 and E7 oncoproteins to reshape epithelial biology, drive and sustain malignant transformation. The high‑risk E7 oncoprotein is multifunctional, and its phosphorylation by Casein Kinase II (CKII) is a key post…
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Candidate 1
- cancerMalignant Cervical Neoplasmdictionary0.60
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