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Clostridium Butyricum and butyrate enhance PD-1 inhibitor therapy in non-small cell lung cancer by regulating the HDAC1/ID2/IL-12R pathway.

Zhuo Chen, Meng-Lei Wang, Zhuo-Wen Zhang, Peng-Cheng Zhang, Qing-Fang Yang, Yu-Jing Weng, Jia-Long Shen, Wenxian Wang, Lan Shao

Translational oncologyOct 1, 2026PMID 42623870doi:10.1016/j.tranon.2026.102972 PMC13524508Journal ArticlepubmedProvenance
Source
PubMed
Retrieved
Sep 12, 2026
Layer
normalized (units and labels harmonized; values unchanged)
Run
ING-PUBMED-20260912-000001
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BACKGROUND: PD-1 blockade has become an important immunotherapeutic strategy, its clinical efficacy in non-small cell lung cancer (NSCLC) remains suboptimal. This study explores the effect and mechanism of sodium butyrate (NaB) and Clostridium butyricum (C. butyricum) combined with PD-1 inhibitors…

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